How your genes shape insulin sensitivity, glucose handling, and fat burning.
Blood sugar control is a balancing act. After you eat, your body has to move glucose out of the blood and into cells; between meals, it has to release stored fuel without letting blood sugar crash. Insulin is the main signal, and "insulin resistance" is the central problem behind type 2 diabetes, prediabetes, and metabolic syndrome. The genes in this report shape your insulin sensitivity set point, how glucose enters tissues, how you store and burn fuel, and how cleanly you switch between feeding and fasting states. Most carry modest effects that are highly responsive to lifestyle.
Worth knowing: Repeated fasting hypoglycemia (especially in children), exercise- or fasting-induced muscle pain with dark urine (suggesting rhabdomyolysis), or a known fatty-acid oxidation disorder require specialist metabolic-genetics care - not self-management.
Answer the symptom questions below as honestly as you can. Combined with your DNA file, your answers help map which pathways are most worth your attention, and which support strategies give you the highest leverage.
Click to choose your DNA file, or drag and drop it here
Accepted: .txt, .csv, .zip— file stays private and is deleted after your report is generated
Once you submit, your personalized PDF report will be generated and emailed to you, typically within a few minutes. Please check your spam folder if it doesn't arrive.
This report is for educational purposes only and is not medical advice. It does not diagnose diabetes or any inherited metabolic disorder. Fasting hypoglycemia, exertional rhabdomyolysis, or known fatty-acid oxidation disorders require specialist care and an emergency sick-day plan. Consult your healthcare provider before changing supplements, medications, or your diet.